OBJECTIVE: To discuss the physiologic mechanisms underlying neurocardiogenic syncope in the context of several different management strategies. The vasovagal reflex in humans appears to have a long evolutionary history, 16 but the basis for the reflex remains uncertain. Syncope is a transient loss of consciousness (TLOC) due to transient global cerebral hypoperfusion characterized by rapid onset, short duration and spontaneous and complete recovery. Both are defined by debilitating symptoms and signs while upright relieved by recumbency. In addition, some children even experience trauma during attacks. While doctors often refer to it as a "simple fainting spell," the mechanism of vasovagal syncope actually is not all that simple.

the person usually regains consciousness and becomes It's generally not a dangerous condition. PATHOPHYSIOLOGY: Neurocardiogenic syncope or the "common faint" is variously called neurally mediated hypotension, vasovagal syncope, or vasodepressor syncope. It happens when the blood vessels open too wide and/or the heartbeat slows, causing a temporary lack of blood flow to the brain. In this regard, several theories have developed, two of which may be based on early protective mechanisms. Pathophysiological mechanisms have remained elusive although our past …

Unlike other causes of fainting, it’s not a sign of a problem with the heart or brain

The cerebrovascular changes that occur prior to vasovagal syncope (VVS) are unclear, with both increases and decreases in cerebrovascular resistance being reported during pre-syncope. Vasovagal Syncope (VVS,simple faint) is the most common cause of transient loss of consciousness and is the acute episodic form of orthostatic intolerance(OI). It has also been known as vasovagal syncope, neurocardiogenic syncope, common or emotional fainting, or reflex syncope. Some authors have hypothesized this type of syncope as a “defense mechanism” for the organism and a few theories have been postulated. 1 The majority of syncope is attributed to simple faint, denoted vasovagal syncope (VVS), 2 and often triggered by upright posture. Vasovagal syncope is the most common cause of fainting. VVS can be typical or non-typical.

It's also called reflex, neurocardiogenic, vasovagal (VVS) or vasodepressor syncope. Many observations suggest that typical (emotional or orthostatic) vasovagal syncope (VVS) is not a disease, but rather a manifestation of a non-pathological trait. The pathophysiology of neurocardiogenic syncope … 5 The most common triggers of vasovagal syncope in older … It's benign and rarely requires medical treatment. It’s the most common cause of fainting. And misunderstanding how vasovagal syncope works can lead to problems in making the correct diagnosis or in choosing adequate treatment. Vasovagal syncope (VVS) is a clinical manifestation of the vasovagal reflex, characterized by the occurrence of bradycardia and hypotension. Neurally mediated syncope (NMS) is the most common form of fainting and a frequent reason for emergency department visits. If you feel lightheaded, nauseous, or sweaty, lie down right away and raise your legs. Therefore, it is particularly important to clarify the pathogenesis of VVS. Vasovagal syncope accounts for more than half of all episodes of syncope.

Vasovagal syncope (VVS) accounts for 60–80% of cases of neurally mediated syncope.

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